Which muscles control breathing




















The process of breathing out called exhalation or expiration is usually passive when a person is not exercising. The elasticity of the lungs and chest wall, which are actively stretched during inhalation, causes them to return to their resting shape and to expel air out of the lungs when inspiratory muscles are relaxed.

Therefore, when a person is at rest, no effort is needed to breathe out. During vigorous exercise, however, a number of muscles participate in exhalation. The abdominal muscles are the most important of these. Abdominal muscles contract, raise abdominal pressure, and push a relaxed diaphragm against the lungs, causing air to be pushed out. The muscles used in breathing can contract only if the nerves connecting them to the brain are intact. In some neck and back injuries, the spinal cord can be severed Injuries of the Spinal Cord and Vertebrae Most spinal cord injuries result from motor vehicle crashes, falls, assaults, and sports injuries.

Symptoms, such as loss of sensation, loss of muscle strength, and loss of bowel, bladder, and Some people with respiratory failure need a mechanical ventilator a machine that helps air get When the diaphragm contracts and moves lower, the chest cavity enlarges, reducing the pressure inside the lungs. To equalize the pressure, air enters the lungs. When the diaphragm relaxes and moves back up, the elasticity of the lungs and chest wall pushes air out of the lungs.

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Common Health Topics. Respiratory muscles. Biology of the Lungs and Airways. Test your knowledge. Coughing up blood from the respiratory tract is called hemoptysis. The brain determines when we need to breathe based on the signals it receives from our organs and nerves. Although we can breathe in and out on command — for example, during pulmonary function testing or when completing breathing exercises — respiratory muscle function is primarily an automatic task.

We have two types of respiratory muscles, inspiratory and expiratory, to accomplish this task. The inspiratory muscles contract to draw air into the lungs. The most important muscle of inspiration is the diaphragm; however, the external intercostals assist with normal quiet breathing. Contraction of the diaphragm increases the space in the thoracic cavity and the lungs fill with air from the external environment.

Accessory muscles of inspiration — sternocleidmastoids, scalenes, serratus, pectoralis — contribute less during normal breathing periods and more during active breathing periods, e. Expiration is a passive process because the lungs naturally want to recoil inward and collapse. During expiration, the lungs deflate without much effort from our muscles. However, the expiratory muscles — internal intercostals, rectus abdominis, external and internal obliques, transversus abdominis — can contract to force air out of the lungs during active breathing periods.

Breathing is a necessary function of life, and most of the time it is effortless. However, if you have lung disease or if you exercise regularly, you may think about breathing more often.

And if the muscles are overloaded or weak because of disease, then breathing may be the only thing you can think about.

When demand increases, the RMs must respond. The initial response is an increase in contraction of the diaphragm and recruitment of accessory muscles. However, if the muscles are weak or functionally overloaded the demand may not be met.

Fatigue of the diaphragm causes a decrease in muscle contraction and alternating movements of the abdomen and rib cage, known as paradoxical breathing. The typical breathing pattern of a person experiencing RM fatigue is rapid and shallow. This type of breathing pattern is not sustainable for long periods of time. Inspiratory tidal volumes are unable delivery fresh oxygen to the alveoli and remove enough carbon dioxide waste.

As the RMs contract more frequently, they require more oxygen and produce more carbon dioxide. The oxygen delivered to the RM gets robbed from other skeletal muscles. So, those muscles begin to experience fatigue. Respiratory muscle training RMT is a way to improve strength or endurance of the inspiratory or expiratory muscles. Strength training is accomplished by breathing against a resistance. Two common examples of resistance strength training include breathing through a small opening flow resistance and breathing around a spring-loaded valve pressure resistance.

In both examples, the resistance load can be adjusted to change the workout, e. Endurance training involves breathing at an above normal respiratory rate and tidal volume for a prolonged period of time, usually 30 minutes. The VIH method requires sophisticated equipment and oversight; therefore, resistance training is used most often when attempting RM training. Focusing specifically on strength training, there are devices available to perform inspiratory muscle training IMT only, expiratory muscle training EMT only, and both inspiratory and expiratory muscle training also known as concurrent respiratory muscle training.

Inspiratory muscle training has received more attention in the medical literature compared to expiratory or concurrent muscle training. There is plenty of literature to support the use of RM training. An Internet search will return a large number of articles, and YouTube can provide videos of device use.

In her book Respiratory Muscle Training: Theory and Practice Elsevier, , she discusses the structural and functional RM changes that occur when using a RM training device, such as increases in diaphragm thickness, changes in muscle fiber type greater fatigue resistance , and improvements in strength.

The benefit extends to athletes in most sports and multiple disease diagnoses. Focusing specifically on COPD, there is a typical pattern that exists. The lungs are overinflated, the diaphragm tends to be flat at rest and the respiratory muscles are functionally and structurally weak.

These changes contribute to complaints of dyspnea — difficulty breathing or breathlessness — during exercise. Dyspnea associated with physical activity can be quantified using the modified Borg Scale. Avoidance of physical activity because of dyspnea can lead to additional skeletal muscle weakness. Respiratory muscle weakness and fatigue are contributing factors to the breathlessness sensation. A stronger muscle can contract more forcefully and potentially resist fatigue. A meta-analysis in Respiratory Medicine conducted by Geddes et al in reported significant improvements in inspiratory muscle strength, inspiratory muscle endurance, exercise capacity, dyspnea and quality of life in COPD patients following IMT.

Complete support that RMT can offer additional benefit over pulmonary rehabilitation PR alone is lacking. It is possible that the lack of support is related to study design and training protocol versus questionable benefits of RMT.

We can test the strength of therespiratory muscles using a basic pressure manometer or sophisticatedequipment. From this test we can determine if the person has RM weaknessusing predicted equations. Obviously, weakermuscles result in a greater benefit from RM training.

Therefore, we can expectto see bigger gains in some individuals. But should we expect the same amountof improvement in someone who is at 40 percent of predicted and someone who isat 75 percent? Also, what training intensity should be used? In general,training intensity should be no lower than 30 percent of the maximum strengthof the muscles.

But what is best? Should we start at 60 percent of the maximumstrength, or 70 percent of the maximum strength? Each GOLD stage indicates a more advanced diseaseprocess. So should ourtraining regimen change based on GOLD stage 1 versus 2, 3 or 4? As always,additional research is needed. And that new research will surely generateadditional questions on the best way to train the respiratory muscles.

A respiratory therapist providesspecialized care for patients with cardiopulmonary disorders.



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